Monday, July 25, 2005
Reason: The Myth of Millions of Years: Creationist cosmology is full of wormholes
"How do you know that you, your memories, and the whole universe with its 'history' weren't called into existence just 5 minutes ago? ":
No time for a lengthy discourse on the absolute fallacy of the hypothesis known as macro evolution (why do they call is a theory when it is more properly labeled as an untested hypothesis?). However, for the record, Macro-Evolultion is the salve that some have replaced religion with however the replacement fails in that it is 1)unproven, 2) posits a reason why life exists without solid proof (like most religions), and 3) requires blind faith and ignorance of illogical and contradictory messages to believe.
"To suppose that the eye, with all its inimitable contrivances for adjusting the focus to different distances, for admitting different amounts of light, and for the correction of spherical and chromatic aberration, could have been formed by natural selection, seems, I freely confess, absurd in the highest possible degree." Charles Darwin, 1859.
He then went on the state that "reason" told him that his theory could support the development of the eye....blah, blah, blah.
Could the eye have evolved from a light sensing cell mass? Maybe, if it gave its mutant carrier an advantage. However, see blood clotting. No way it could have evolved unless every aspect of it spontaneously "evolved"/mutated in one fell swoop: Not part of the theory of evollution but rather creationism.
There's the rub, evolutionary theory doesn't allow for instantaneous macro-evolutionary changes and irreducible complexity like clotting doesn't buttress Mr. Darwin and his believers.
The Blood Clotting Cascade :
1. A cut occurs and Hageman Factor sticks to the surface of cells near the wound. Bound Hageman Factor reacts with another enzyme called HMK to produce Activated Hageman.
2. Pre Kallikrein reacts with Activated Hageman to produce Kallikrein.
3. Hageman Factor also reacts with HMK and Kallikrein to form Activated Hageman.
4. PTA reacts with Activated Hageman and HMK to produce Activated PTA.
5. Christmas Factor reacts with Activated PTA and Convertin to produce Activated Christmas Factor.
6. Antihemophilic Factor is activated by Thrombin to produce Activated Antihemophilic Factor.
7. Stuart Factor reacts with Activated Christmas Factor and Activated Antihemophilic Factor to produce Activated Stuart Factor.
8. Proconvertin is activated by Activated Hageman Factor to produce Convertin.
9. When a cut occurs, Tissue Factor (which is only found outside of cells) is brought in near the wound where it reacts with Convertin and Stuart Factor to produce Activated Stuart Factor. (Note that step 9 involves an extrinsic process whereas step 7 is an intrinsic process.)
10. Proaccelerin is activated by Thrombin to produce Accelerin.
11a. GLU-Prothrombin reacts with Prothrombin Enzyme and Vitamin K to produce GLA-Prothrombin. (Note that Prothrombin cannot be activated in the GLU form so it must be formed into the GLA form. In this process ten amino acids must be changed from glutamate to gama carboxy glutamate.)
11b. GLS-Prothrombin is then able to bind to Calcium. This allows GLA-Prothrombin to stick to surfaces of cells. Only intact modified Calcium-Prothrombin Complex can bind to the cell membrane and be cleaved by Activated Stuart and Accerlerin to produce Thrombin.
12. Prothrombin-Ca (bound to cell surface) is activated by Activated Stuart to produce Thrombin.
13.Prothrombin also reacts with Activated Stuart and Accelerin to produce Thrombin. (Step 13 is much faster than step 12.)
14. Fibrinogin is activated by Thrombin to produce Fibrin. Threads of Fibrin are the final clot. However, it would be more effective if the Fibrin threads could form more cross links with each other.
15. FSF (Fibrin Stabilizing Factor) is activated by Thrombin to form Activated FSF.
16. When Fibrin reacts with Activated FSF many more cross ties are made with other Fibrin filaments to form a more effective clot.
Any mutation that gave rise to one of these enzymes or even several at one time would have rendered the mutant unfit comparatively. Nature doesn't say "wow, this mutant being may be able to make its life-blood clot if it can reproduce and continue to spontaneously evolve," and thus make sure that it lives to spawn. No, this mutant dies because it expends more calories than its counterparts making the unecessary enzymes and never gets to reproduce because it probably starves. Darwinism....what a crock.
No time for a lengthy discourse on the absolute fallacy of the hypothesis known as macro evolution (why do they call is a theory when it is more properly labeled as an untested hypothesis?). However, for the record, Macro-Evolultion is the salve that some have replaced religion with however the replacement fails in that it is 1)unproven, 2) posits a reason why life exists without solid proof (like most religions), and 3) requires blind faith and ignorance of illogical and contradictory messages to believe.
"To suppose that the eye, with all its inimitable contrivances for adjusting the focus to different distances, for admitting different amounts of light, and for the correction of spherical and chromatic aberration, could have been formed by natural selection, seems, I freely confess, absurd in the highest possible degree." Charles Darwin, 1859.
He then went on the state that "reason" told him that his theory could support the development of the eye....blah, blah, blah.
Could the eye have evolved from a light sensing cell mass? Maybe, if it gave its mutant carrier an advantage. However, see blood clotting. No way it could have evolved unless every aspect of it spontaneously "evolved"/mutated in one fell swoop: Not part of the theory of evollution but rather creationism.
There's the rub, evolutionary theory doesn't allow for instantaneous macro-evolutionary changes and irreducible complexity like clotting doesn't buttress Mr. Darwin and his believers.
The Blood Clotting Cascade :
1. A cut occurs and Hageman Factor sticks to the surface of cells near the wound. Bound Hageman Factor reacts with another enzyme called HMK to produce Activated Hageman.
2. Pre Kallikrein reacts with Activated Hageman to produce Kallikrein.
3. Hageman Factor also reacts with HMK and Kallikrein to form Activated Hageman.
4. PTA reacts with Activated Hageman and HMK to produce Activated PTA.
5. Christmas Factor reacts with Activated PTA and Convertin to produce Activated Christmas Factor.
6. Antihemophilic Factor is activated by Thrombin to produce Activated Antihemophilic Factor.
7. Stuart Factor reacts with Activated Christmas Factor and Activated Antihemophilic Factor to produce Activated Stuart Factor.
8. Proconvertin is activated by Activated Hageman Factor to produce Convertin.
9. When a cut occurs, Tissue Factor (which is only found outside of cells) is brought in near the wound where it reacts with Convertin and Stuart Factor to produce Activated Stuart Factor. (Note that step 9 involves an extrinsic process whereas step 7 is an intrinsic process.)
10. Proaccelerin is activated by Thrombin to produce Accelerin.
11a. GLU-Prothrombin reacts with Prothrombin Enzyme and Vitamin K to produce GLA-Prothrombin. (Note that Prothrombin cannot be activated in the GLU form so it must be formed into the GLA form. In this process ten amino acids must be changed from glutamate to gama carboxy glutamate.)
11b. GLS-Prothrombin is then able to bind to Calcium. This allows GLA-Prothrombin to stick to surfaces of cells. Only intact modified Calcium-Prothrombin Complex can bind to the cell membrane and be cleaved by Activated Stuart and Accerlerin to produce Thrombin.
12. Prothrombin-Ca (bound to cell surface) is activated by Activated Stuart to produce Thrombin.
13.Prothrombin also reacts with Activated Stuart and Accelerin to produce Thrombin. (Step 13 is much faster than step 12.)
14. Fibrinogin is activated by Thrombin to produce Fibrin. Threads of Fibrin are the final clot. However, it would be more effective if the Fibrin threads could form more cross links with each other.
15. FSF (Fibrin Stabilizing Factor) is activated by Thrombin to form Activated FSF.
16. When Fibrin reacts with Activated FSF many more cross ties are made with other Fibrin filaments to form a more effective clot.
Any mutation that gave rise to one of these enzymes or even several at one time would have rendered the mutant unfit comparatively. Nature doesn't say "wow, this mutant being may be able to make its life-blood clot if it can reproduce and continue to spontaneously evolve," and thus make sure that it lives to spawn. No, this mutant dies because it expends more calories than its counterparts making the unecessary enzymes and never gets to reproduce because it probably starves. Darwinism....what a crock.
# posted by W. : 1:43 PM
